Whole-body 18F-FDG PET/CT scan in a patient with Pancreatic Cancer taken from the PETWB-REP dataset. The following English report (translated from original Chinese) is taken verbatim from the public dataset and has not been modified or otherwise checked for accuracy (see the end for citation).
Findings
After fasting and intravenous injection of 18F-FDG, a whole-body PET/CT scan was performed.
The whole-body scan showed: A few punctate low-density shadows were seen in the deep bilateral cerebral regions; no abnormal density shadows were seen in the remaining brain parenchyma.
FDG uptake was normal.
The ventricles, sulci, fissures, and cisterns were widened; the ventricles were symmetrical, and there was no midline shift.
The eyeballs were symmetrical bilaterally, with no obvious abnormalities.
The paranasal sinuses showed no thickening of the mucosa, and the sinus walls were intact.
The nasopharyngeal wall showed no thickening, and FDG uptake was normal.
The pharyngeal recesses were symmetrical bilaterally, the Eustachian tube openings were not narrowed, the infratemporal and pterygopalatine fossae were structurally normal, and the bilateral parapharyngeal spaces were clear with no abnormal FDG uptake.
The palatine tonsils showed physiological uptake.
No abnormal density shadows were seen in the bilateral parotid and submandibular glands.
The laryngopharynx showed no abnormalities in morphology or structure.
Focal FDG uptake was observed in the tongue, with an SUVmax of 7.6 and an uptake area of approximately 2.0 1.5 cm.
The thyroid gland was normal in shape and size, but its density was somewhat heterogeneous; FDG uptake was not abnormal.
No enlarged lymph nodes were observed in the bilateral deep cervical spaces or submandibular region.
Several small ground-glass nodules were observed in both lungs, the largest located in the anterior segment of the right upper lobe, with a CT value of approximately -523 HU, a long diameter of approximately 0.7 cm, and relatively clear borders; FDG uptake was not abnormal.
Scattered linear opacities and calcifications were observed in the remaining lungs; FDG uptake was not abnormal in any of these areas.
No pleural thickening was observed bilaterally, and there was no pleural effusion or pneumothorax.
No significantly enlarged lymph nodes were observed in the bilateral hilar and mediastinal regions.
The cardiac silhouette was full.
Calcification was observed in some arterial walls (including the coronary arteries).
No esophageal dilatation, significant wall thickening, or mass was observed; FDG uptake was not increased.
Bilateral breasts are normal, and FDG metabolism is normal.
The liver's shape and size are normal, with smooth borders and no widening of the hepatic fissure.
A low-density nodule, approximately 1.5 cm in length, is seen in the second hepatic hilum, with increased FDG metabolism (SUVmax = 5.8).
Multiple low-density lesions are seen within the liver, the largest located at the top of the diaphragm, approximately 1.0 cm in length, with no abnormal FDG uptake.
The main portal vein is not significantly widened, and no dilation is seen in the intrahepatic or extrahepatic bile ducts.
The gallbladder is absent post-operatively.
An irregular low-density mass, approximately 4.4 3.8 cm in size, is seen in the head and neck of the pancreas, with uneven density and increased FDG metabolism (SUVmax = 7.0).
The main pancreatic duct is dilated to varying degrees, and the pancreatic parenchyma in the body and tail is atrophied.
A localized low-density nodule, approximately 1.7 cm in length, is seen in the pancreatic body, with no abnormal FDG metabolism.
Multiple flocculent shadows, soft tissue nodules, and masses were observed in the peritoneum, the largest measuring approximately 3.1 2.0 cm, located in the right paracolic gutter.
FDG metabolism was increased, with an SUVmax of 8.3.
The spleen showed no abnormalities in morphology, size, density, or FDG uptake.
Both kidneys were normal in shape and size, with no obvious abnormal density shadows in the parenchyma.
The renal pelvis, calyces, and ureters were not widened, and FDG uptake was not significantly abnormal.
Bilateral adrenal glands showed no obvious abnormalities on contrast imaging.
The stomach was poorly distended, with localized thickening and dense shadows in the gastric fundus wall, measuring approximately 2.2 1.5 cm, showing increased FDG metabolism (SUVmax = 4.3).
Continuous FDG metabolism was also observed in the colon and rectum (SUVmax = 6.9).
The uterus had a normal shape and no abnormal FDG metabolism.
No abnormal FDG metabolism was observed in the bilateral adnexa.
The bladder is generally full, with no obvious positive stones.
An enlarged lymph node, approximately 0.9 cm in short diameter, is seen anterior to the left aorta, showing increased FDG metabolism (SUVmax = 5.9).
A small amount of pelvic effusion is present.
The spinal alignment is normal, with some vertebral body margin osteophytes and L4/5 and L5/S1 disc bulges.
Osteophytes are present in the right temporomandibular joint, obscuring the joint space; FDG uptake is normal.
Systemic bone marrow FDG metabolism is normal.
Impression
a. Mass in the head and neck of the pancreas, with increased FDG metabolism, suggestive of malignancy, possibly pancreatic cancer or intraductal papillary tumor of the pancreas. Please correlate with clinicopathology. b. Lymph node metastasis to the left anterior aorta. Peritoneal seeding metastasis. Possible metastasis to the second hepatic hilum. Please correlate with contrast-enhanced MRI. Small amount of pelvic effusion. c. Localized low-density nodule in the pancreatic body, with normal FDG metabolism, suggestive of a cyst.
Localized thickening and dense shadow in the gastric fundus wall, with increased FDG metabolism, tumor cannot be ruled out. Gastroscopy is recommended.
Multiple low-density lesions in the liver, with normal FDG uptake, suggestive of hemangioma or cyst.
Several ground-glass nodules in both lungs, suggestive of inflammatory nodules or atypical adenomatous hyperplasia. Annual HRCT follow-up is recommended. Scattered chronic inflammation and old lesions in both lungs. Cardiac shadow is full. Calcification of some arterial walls (including coronary arteries).
Continuous increased FDG metabolism in the colon and rectum, suggestive of inflammation or physiological uptake.
Focal increased FDG metabolism in the tongue, likely physiological; clinical correlation is recommended.
Degenerative changes in the spine. L4/5 and L5/S1 intervertebral disc bulges. Benign osteopathy of the right temporomandibular joint.
A few ischemic lesions in the deep bilateral brain, suggestive of age-related encephalopathy.
This case is from PETWB-REP, a curated dataset of whole-body 18F-FDG PET/CT scans and corresponding radiology reports from 490 patients with a broad spectrum of malignancies. The data were retrospectively collected from patients who underwent clinically indicated whole-body 18F-FDG PET/CT scans at the Shanghai Universal Medical Imaging Diagnostic Center between 2021 and 2024.
License: Creative Commons Attribution 4.0 International (CC BY 4.0)
Citation:
Xue, L., Feng, G., Wenbo, Z., Zhang, Y., Li, L., Wang, S., Peng, L., Peng, S., & Gao, X. (2026). PETWB-REP: A Multi-Cancer Whole-Body FDG PET/CT Dataset with Corresponding Radiology Reports [Data set]. Zenodo. https://doi.org/10.5281/zenodo.18670487
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