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Whole-body 18F-FDG PET/CT scan in a patient with Liver Cancer taken from the PETWB-REP dataset. The following English report (translated from original Chinese) is taken verbatim from the public dataset and has not been modified or otherwise checked for accuracy (see the end for citation).

Findings

After fasting and intravenous injection of 18F-FDG, a whole-body PET/CT scan was performed.
The whole-body scan showed: a few punctate low-density shadows in the deep bilateral cerebral regions; no abnormal density shadows were seen in the remaining brain parenchyma, and FDG uptake was not significantly abnormal.
The ventricles, sulci, fissures, and cisterns were widened, with symmetrical bilateral ventricles and no midline shift.
Both eyeballs were symmetrical and showed no obvious abnormalities.
The paranasal sinuses showed no thickening of the mucosa, and the sinus walls were intact.
The nasopharyngeal wall showed no thickening, and FDG uptake was not abnormal.
The bilateral pharyngeal recesses were symmetrical, the Eustachian tube openings were not narrowed, the infratemporal and pterygopalatine fossae were structurally normal, and the bilateral parapharyngeal spaces were clear, with no abnormal FDG uptake.
The bilateral palatine tonsils showed physiological uptake.
The laryngopharynx showed no abnormalities in morphology or structure.
The bilateral parotid and submandibular glands showed no abnormal density shadows.
The thyroid gland was normal in shape and size, with uniform density, and FDG uptake was not abnormal.
Multiple scattered patchy and linear shadows were seen in both lungs, with no abnormal FDG metabolism.
Small amount of pleural effusion in both pleural cavities.
Several lymph nodes are seen in the bilateral hilum, pretracheal space, aortic window, and below the carina, the largest being approximately 1.0 cm in short diameter, with increased FDG metabolism (SUVmax = 3.5).
Cardiac silhouette is normal.
Esophageal dilation is not seen, nor is there significant thickening or mass in the esophageal wall; FDG uptake is not increased.
Bilateral breasts are normal, with no abnormal FDG metabolism.
Liver outline is irregular; multiple irregular dense shadows are seen in the intrahepatic bile ducts, common bile duct, and gallbladder, the largest being approximately 2.6 cm in long diameter, with no abnormal FDG metabolism.
An irregular low-density mass is seen in the left lobe of the liver, approximately 6.0 3.6 cm in size, with increased FDG metabolism (SUVmax = 10.2); intrahepatic bile duct dilation is also present.
Multiple irregular soft tissue nodules are seen in the subcapsular region, mesentery, left paracolic gutter, pelvic floor peritoneum, and right inguinal canal, with increased FDG metabolism (SUVmax = 4.9).
Multiple enlarged lymph nodes were observed in the porta hepatis, hepatogastric space, para-aortic region, peripancreatic region, bilateral inguinal regions, bilateral internal mammary chains, and left supraclavicular fossa.
The largest lymph node had a short diameter of approximately 1.8 cm, with increased FDG metabolism (SUVmax = 5.9).
Abdominal and pelvic effusion was present.
The pancreas was normal in shape, with no obvious abnormal density shadows in the parenchyma.
The main pancreatic duct was not widened, and FDG uptake was not significantly abnormal.
The spleen showed no abnormalities in shape, size, density, or FDG uptake.
Both kidneys were normal in shape and size, with no obvious abnormal density shadows in the parenchyma.
The renal pelvis, calyces, and ureters were not widened, and FDG uptake was not significantly abnormal.
Bilateral adrenal glands showed no significant abnormalities on contrast.
The stomach was poorly distended, with no significant thickening of the gastric wall and no significant abnormalities in FDG uptake.
The intestines were poorly distended, with increased FDG metabolism in some intestinal segments (SUVmax = 5.9).
The uterus was not clearly visualized.
The bladder was generally full, with no obvious positive stones observed.
Kyphosis of the spine, compression and flattening of some thoracic and lumbar vertebrae, osteophyte formation at the margins of some vertebral bodies, and L4/5 and L5/S1 intervertebral disc bulging.
No abnormalities were observed in FDG uptake.
No abnormal FDG metabolism was observed in the entire skeleton.

Impression

  1. a. A mass in the left lobe of the liver with elevated FDG metabolism, suggestive of malignancy, most likely intrahepatic cholangiocarcinoma. b. Multiple lymph node metastases in the abdominal cavity, retroperitoneum, bilateral inguinal regions, bilateral internal mammary chains, and left supraclavicular fossa. Extensive seeding metastases in the abdominoperineal cavity and right inguinal canal. Abdominal and pelvic effusion. c. Multiple stones in the intrahepatic bile ducts, common bile duct, and gallbladder.

  2. Scattered chronic inflammation and fibrosis in both lungs. Small amount of pleural effusion bilaterally. Reactive hyperplasia of hilar and mediastinal lymph nodes bilaterally.

  3. Elevated FDG metabolism in some intestinal segments, suggestive of inflammatory or physiological uptake.

  4. Kyphosis of the spine, with some old compression fractures of the thoracic and lumbar vertebrae. Spinal degeneration. L4/5 and L5/S1 intervertebral disc bulges.

  5. A few ischemic lesions in the deep bilateral cerebral regions, suggestive of age-related brain changes.

This case is from PETWB-REP, a curated dataset of whole-body 18F-FDG PET/CT scans and corresponding radiology reports from 490 patients with a broad spectrum of malignancies. The data were retrospectively collected from patients who underwent clinically indicated whole-body 18F-FDG PET/CT scans at the Shanghai Universal Medical Imaging Diagnostic Center between 2021 and 2024.

License: Creative Commons Attribution 4.0 International (CC BY 4.0)

Citation:
Xue, L., Feng, G., Wenbo, Z., Zhang, Y., Li, L., Wang, S., Peng, L., Peng, S., & Gao, X. (2026). PETWB-REP: A Multi-Cancer Whole-Body FDG PET/CT Dataset with Corresponding Radiology Reports [Data set]. Zenodo. https://doi.org/10.5281/zenodo.18670487

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