Whole-body 18F-FDG PET/CT scan in a patient with Lung Cancer taken from the PETWB-REP dataset. The following English report (translated from original Chinese) is taken verbatim from the public dataset and has not been modified or otherwise checked for accuracy (see the end for citation).
Findings
After fasting and intravenous injection of 18F-FDG, and resting, a whole-body PET/CT scan was performed.
The whole-body scan showed: A round soft tissue mass, approximately 3.1 3.6 cm in size, was visible in the posterior segment of the right upper lobe.
It was superficially lobulated with spiculated margins, and showed pleural thickening due to traction.
Vascular penetration and bronchial truncation were visible.
The density was uneven, and punctate calcifications were visible within the mass.
The average CT value was approximately 42 HU.
FDG metabolism was increased, with an SUVmax of 12.8.
Some adjacent bronchi were compressed and narrowed.
Multiple nodules, approximately 0.2?.5 cm in diameter, were visible in both lungs.
FDG metabolism was not increased.
Multiple calcifications were visible in both lungs.
Increased translucency was observed in the upper lobes of both lungs, with diffusely distributed air-filled cavities.
The largest lesion in the anterior segment of the right upper lobe had a long diameter of approximately 0.9 cm and small patchy blurred shadows at the edges.
Scattered linear and punctate shadows were present in both lungs, with some adjacent pleural thickening.
Several lymph nodes were observed in the right hilum, aortic window, and below the carina, the largest being located in the right hilum, with a short diameter of approximately 1.0 cm.
FDG metabolism was increased, with an SUVmax of 3.7.
Calcifications were visible in the aorta and its branches (including the coronary arteries).
Osteolytic destruction was observed at the anterior margin of the S1 vertebral body and the right femoral head, the latter being larger, measuring approximately 1.9*4.2 cm, with increased FDG metabolism and an SUVmax of 7.1.
A few punctate low-density shadows were seen in the deep bilateral cerebral regions, with no significant abnormalities in FDG uptake.
No widening of the ventricles, sulci, fissures, or cisterns was observed, and local density and FDG metabolism were normal.
Midline shift was not observed.
The bilateral eyeballs showed normal morphology and outline, with clear retrobulbar structures and normal FDG metabolism.
No significant thickening of the paranasal sinus mucosa was observed, and the sinus walls were intact.
Poor pneumatization of the bilateral mastoid air cells was observed.
No significant thickening of the soft tissue on both sides of the nasopharynx was observed; the pharyngeal recesses were symmetrical; and FDG metabolism was normal.
The palatine tonsils were full bilaterally, with normal FDG uptake.
The morphology and structure of the laryngopharynx were normal; the parapharyngeal spaces were clear.
The size, shape, and density of the bilateral submandibular glands were normal; and FDG uptake was normal.
The density of the bilateral parotid glands was normal; FDG metabolism was increased, with SUVmax = 2.8.
A low-density nodule was observed in the left lobe of the thyroid gland, with a long diameter of approximately 0.8 cm; FDG metabolism was normal.
Small lymph nodes were visible in the bilateral deep cervical spaces, submandibular region, and submental region; the largest was approximately 0.5 cm in the short diameter of the left deep cervical space; FDG uptake was normal.
The esophagus was not dilated; the esophageal wall was not significantly thickened or swollen; and FDG metabolism was normal.
The stomach was poorly filled; no obvious mass was observed; and FDG metabolism was normal.
Intestinal distension was unsatisfactory; no local masses were observed; FDG uptake was normal in some intestinal segments.
The liver showed no obvious abnormalities in shape or size; the liver margins were smooth; and the hepatic fissures were not widened.
No obvious abnormal density shadows were observed in the liver parenchyma; FDG metabolism was normal.
No dilation was observed in the intrahepatic or extrahepatic bile ducts.
The gallbladder showed no abnormalities in shape or size; the gallbladder wall was not thickened; no positive stones or obvious masses were observed; FDG metabolism in the gallbladder fossa was normal.
The peripancreatic spaces were clear; no obvious abnormal density shadows were observed in the parenchyma; the pancreatic duct was not widened; and FDG metabolism was normal.
The spleen was basically normal in shape and size; density and FDG metabolism were normal.
The bilateral adrenal glands showed no abnormalities in shape, size, or density; local FDG metabolism was normal.
A cystic low-density nodule approximately 2.0 cm in diameter is visible in the right kidney, with absent FDG metabolism.
The left kidney is normal in shape and size, with no obvious abnormal density shadows in the renal parenchyma, and no obvious abnormal FDG metabolism.
The mid-segment of the left ureter is locally widened, approximately 1.3 cm, with a smooth wall and no obvious abnormal density shadows; FDG metabolism is normal.
No widening is seen in the bilateral renal pelvis and calyces, or the right ureter; no positive stones are observed locally.
The bladder is adequately filled, with no obvious localized thickening or mass in the wall, and no positive stones are seen within the lumen.
The prostate is normal in shape and size, with punctate calcifications observed internally; FDG metabolism is normal.
No significantly enlarged lymph nodes are seen in the retroperitoneum, pelvis, or bilateral inguinal regions.
No significant effusion is seen in the abdominal or pelvic cavities.
The spinal alignment is normal, with osteophyte formation at some vertebral body margins and facet joints.
FDG metabolism is normal.
Low-density gas shadows are visible within the L4/5 and L5/S1 intervertebral discs, with disc bulging outwards and compression of the dural sac.
Impression
a. A mass in the posterior segment of the right upper lobe, with increased FDG metabolism, suggestive of lung cancer; please correlate with clinicopathology. b. Bone destruction at the anterior margin of the S1 vertebral body and the right femoral head with increased FDG metabolism, suggestive of metastasis; please correlate with MRI. c. Possible reactive hyperplasia of the right hilar and mediastinal lymph nodes; partial metastasis to be ruled out. d. Bilateral emphysema with bullous formation; bullous formation with minor inflammation in the anterior segment of the right upper lobe. Multiple chronic inflammatory nodules in both lungs; CT follow-up recommended. Minor chronic inflammation and sequelae (including calcifications) in both lungs; some adjacent pleural thickening. Partial arteriosclerosis (including coronary arteries).
Low-density nodule in the left lobe of the thyroid gland, highly suggestive of adenoma; please correlate with ultrasound.
Right renal cyst. Dilatation of the mid-segment of the left ureter. Prostatic calcification.
Degenerative changes in the spine. L4/5 and L5/S1 intervertebral disc degeneration and bulging.
A few ischemic lesions in the deep bilateral brain regions; MRI is recommended.
Bilateral mastoid hypopneumatization. Increased FDG metabolism in the bilateral parotid glands, suggestive of physiological uptake or chronic inflammation. Reactive hyperplasia of small cervical lymph nodes bilaterally.
This case is from PETWB-REP, a curated dataset of whole-body 18F-FDG PET/CT scans and corresponding radiology reports from 490 patients with a broad spectrum of malignancies. The data were retrospectively collected from patients who underwent clinically indicated whole-body 18F-FDG PET/CT scans at the Shanghai Universal Medical Imaging Diagnostic Center between 2021 and 2024.
License: Creative Commons Attribution 4.0 International (CC BY 4.0)
Citation:
Xue, L., Feng, G., Wenbo, Z., Zhang, Y., Li, L., Wang, S., Peng, L., Peng, S., & Gao, X. (2026). PETWB-REP: A Multi-Cancer Whole-Body FDG PET/CT Dataset with Corresponding Radiology Reports [Data set]. Zenodo. https://doi.org/10.5281/zenodo.18670487
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