Whole-body 18F-FDG PET/CT scan in a patient with Liver Cancer taken from the PETWB-REP dataset. The following English report (translated from original Chinese) is taken verbatim from the public dataset and has not been modified or otherwise checked for accuracy (see the end for citation).
Findings
After fasting and intravenous injection of 18F-FDG, and resting, a whole-body PET/CT scan was performed.
The whole-body tomographic images showed:The brain morphology and structure were normal, with no abnormal density shadows in the brain parenchyma, and no abnormal FDG uptake.
No widening of the ventricles, sulci, fissures, or cisterns was observed, with no abnormalities in local density or FDG uptake, and no midline shift.
The morphology and outline of both eyeballs were normal, with clear retrobulbar structures, and no abnormal FDG uptake.
The left nasal cavity was enlarged, with partial loss of the left ethmoid sinus and middle turbinate; the right ethmoid sinus and maxillary sinus showed slight local mucosal thickening, and no abnormal FDG uptake.
No significant thickening of the soft tissue on both sides of the nasopharyngeal walls was observed; the bilateral pharyngeal recesses were symmetrical, and no abnormal FDG uptake was seen.
The morphology and structure of the oropharynx and laryngopharynx were normal, and the parapharyngeal spaces were clear.
The parotid and submandibular glands were normal in size, shape, and density, and FDG uptake was physiological.
The thyroid gland was normal in shape and size, but its density was slightly uneven; FDG uptake was normal.
No significantly enlarged lymph nodes were observed in the bilateral deep cervical spaces, submandibular region, or submental region; FDG uptake was normal.
A small solid nodule approximately 0.2 cm in diameter with clear borders was observed in the apical segment of the right upper lobe; FDG uptake was normal.
No significant thickening of the pleura was observed bilaterally, and no significant pleural effusion was observed bilaterally.
No significantly enlarged lymph nodes were observed bilaterally in the hilum and mediastinum; FDG uptake was not significantly increased.
The heart size was normal.
Calcification was observed in the walls of the aorta and its branches.
The lower esophagus wall was slightly thickened; FDG uptake was increased, SUVmax = 2.3.
The stomach was poorly filled, with slight thickening of the stomach wall in some areas.
FDG uptake was increased, with SUVmax = 3.2.
Intestinal filling was also unsatisfactory, with increased FDG uptake in some intestinal segments (SUVmax = 8.8).
After delayed scanning, changes in intestinal morphology and position were observed, with some segments still showing high FDG uptake (SUVmax = 11.7).
The liver was disproportionately large, with irregular borders and slightly widened fissures.
Multiple low-density nodules and masses were seen in the right lobe and left medial lobe of the liver, with indistinct borders.
Some nodules merged into clusters, protruding beyond the liver outline.
The largest fused nodule measured approximately 11.9 8.3 8.1 cm, exhibiting heterogeneous density, containing patchy low-density lesions and slightly high-density shadows.
FDG uptake was at background levels (SUVmax = 3.7).
Delayed scanning did not show further increase in FDG uptake.
The adjacent right branch of the portal vein and the right and middle hepatic veins were not clearly visualized.
The main portal vein is slightly widened, with an anteroposterior diameter of approximately 1.2 cm.
Multiple tortuous, linear soft tissue density shadows are seen around the stomach.
Multiple lymph nodes are visible in the hepatogastric space, porta hepatis, and retroperitoneum, the largest with a short diameter of approximately 0.7 cm; FDG uptake is normal.
Fluid accumulation is present around the liver, spleen, bilateral paracolic gutter, mesentery, and pelvis.
Peritoneal thickening is observed in the perihepatic, splenic, and bilateral paracolic gutter areas, with increased FDG uptake (SUVmax = 2.0).
No dilation of intrahepatic or extrahepatic bile ducts is observed.
The gallbladder is normal in shape and size, with no thickening of the gallbladder wall, no positive stones or obvious masses, and FDG uptake in the gallbladder fossa is normal.
The peripancreatic space is clear, with no obvious abnormal density shadows in the parenchyma; the pancreatic duct is not widened, and FDG uptake is normal.
The spleen is generally normal in shape and size, with no abnormal density or FDG uptake.
The bilateral adrenal glands showed no abnormalities in shape, size, or density, and local FDG uptake was normal.
Both kidneys were normal in shape and size, with no obvious abnormal density shadows in the renal parenchyma, and no obvious abnormalities in FDG uptake.
No widening of the renal pelvis, calyces, or ureters was observed, and no positive stones were seen locally.
The prostate gland showed no abnormalities in shape or size, and no focal abnormal increases in FDG uptake were observed.
Bladder filling was poor, and no positive stones were seen within the bladder.
Spinal alignment was unstable, with osteophyte formation at the margins of some vertebral bodies, slight posterior displacement of the L4 vertebral body, and L4/5 intervertebral disc herniation; no abnormal FDG uptake was observed.
Impression
a. Multiple lesions in the right lobe and left inner lobe of the liver, some fused, with FDG background metabolism, combined with the enhanced MRI report from another hospital, strongly suggest primary liver cancer. b. Multiple lymph nodes in the hepatogastric space, porta hepatis, and retroperitoneum, with no increased FDG metabolism, suggest possible reactive lymph node hyperplasia, with partial metastasis not ruled out. Peripheral thickening around the liver, spleen, and bilateral paracolic gutter, with slightly increased FDG metabolism, suggests possible peritoneal seeding metastasis. Please follow up clinically. c. Liver cirrhosis, portal hypertension with collateral circulation, and abdominopelvic effusion.
Chronic inflammatory changes in the lower esophagus, stomach, and part of the intestine; endoscopic re-examination is necessary if needed.
Chronic inflammatory micronodules in the apical segment of the right upper lobe of the lung. Partial arteriosclerosis.
Degenerative changes in the spine, slight posterior slippage of the L4 vertebral body, and L4/5 disc herniation.
No obvious abnormalities were found on cranial scintigraphy. Postoperative changes in the left nasal cavity, and minor chronic inflammation in the right ethmoid and maxillary sinuses.
This case is from PETWB-REP, a curated dataset of whole-body 18F-FDG PET/CT scans and corresponding radiology reports from 490 patients with a broad spectrum of malignancies. The data were retrospectively collected from patients who underwent clinically indicated whole-body 18F-FDG PET/CT scans at the Shanghai Universal Medical Imaging Diagnostic Center between 2021 and 2024.
License: Creative Commons Attribution 4.0 International (CC BY 4.0)
Citation:
Xue, L., Feng, G., Wenbo, Z., Zhang, Y., Li, L., Wang, S., Peng, L., Peng, S., & Gao, X. (2026). PETWB-REP: A Multi-Cancer Whole-Body FDG PET/CT Dataset with Corresponding Radiology Reports [Data set]. Zenodo. https://doi.org/10.5281/zenodo.18670487
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