MR images more clearly depict the cortical and subcortical T2 hyperintensity with a posterior predominance (@Key Finding 1). The areas of signal abnormality demonstrated diffusion facilitation and not restriction as seen on ADC and are therefore, inconsistent with regions of acute infarction (@Key Finding 2). Note the better seen involvement of the bilateral caudate, medial thalami, and cerebella (@Key Finding 3). Patient symptoms as well as the signal abnormalities on imaging were reversible and resolved, corroborating the diagnosis of PRES.